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mechanism the authors consider the modulation of osteoclastogenesis. A total of 390 ocular video clips were reviewed by 13 emergency physicians. Overall, physicians were able to accurately diagnose the presence of a RD 74.6% (95%CI, 69.8–79.4) of the time, PVD 85.7% (95%CI, 77.6–93.8) of the time, and normal ultrasounds 94.9% (95%CI 87.3–100.0) of the time. There was no statistically significant relationship between correct diagnoses for ocular abnormalities or normal ultrasound images and number of previous ocular ultrasounds performed by emergency physicians.. On the other hand, leptin directly correlates with adiposity (127). Devlin MJ (2011) discusses the key role of leptin in regulating bone marrow fat deposition during starvation (22). Studies have found high amounts of marrow fat in ob/ob mice lacking leptin and db/db mice lacking leptin receptors, irrespective of obesity (22). However, leptin treatment in ob/ob mice was shown to reduce bone marrow fat (326-328). Lower leptin levels lead to a persistence of bone marrow fat, because it promotes autophagy by inhibiting the mTOR protein (329,330). The mTOR protein has shown to inhibit autophagy and promote lipogenesis (329,330). Furthermore, bone marrow fat is resistant to lipolysis until depletion of other fat stores occur (22). Syed et al. (2008) have also found high levels of bone marrow fat in post-menopausal women, suggesting that low estrogen levels are associated with high bone marrow fat (331). In conclusion, these experiments highlight the mechanism through which starvation triggers bone marrow fat deposition (22).

On the other hand, leptin directly correlates with adiposity (127). Devlin MJ (2011) discusses the key role of leptin in regulating bone marrow fat deposition during starvation (22). Studies have found high amounts of marrow fat in ob/ob mice lacking leptin and db/db mice lacking leptin receptors, irrespective of obesity (22). However, leptin treatment in ob/ob mice was shown to reduce bone marrow fat (326-328). Lower leptin levels lead to a persistence of bone marrow fat, because it promotes autophagy by inhibiting the mTOR protein (329,330). The mTOR protein has shown to inhibit autophagy and promote lipogenesis (329,330). Furthermore, bone marrow fat is resistant to lipolysis until depletion of other fat stores occur (22). Syed et al. (2008) have also found high levels of bone marrow fat in post-menopausal women, suggesting that low estrogen levels are associated with high bone marrow fat (331). In conclusion, these experiments highlight the mechanism through which starvation triggers bone marrow fat deposition (22).. improve antitumor efficacy, Shobana et al. [81] have engineered the

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